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Podcast 483: Feral Cells

In this episode, we examine the concept of feral tissue, exploring its fundamental triggers—Short Chain Fatty Acids, Aryl Hydrocarbon Receptors, and pH imbalance. We discuss Spencer Feldman’s innovative, research-oriented protocol aimed at addressing feral tissue growth through a comprehensive 6-step program. This protocol includes products designed to repair the microbiome with short chain fatty acids and lactic acid. As well as products to shift pH balance. It also employs a trophoblast program using pancreatic enzymes and electrons. While promising, these methods are currently unproven and intended solely for research purposes. They are not medical advice, nor have they been proven safe or effective.

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MARTIN: Well, hello, it’s Martin Pytela. Life Enthusiast podcast. And with me Spencer Feldman Remedy Link Chief Formulator. Today we have a topic that is big. In the sense that a lot of people end up with very tragic health outcomes when they mismanage what we’re going to talk about today. Anyway, Spencer, let’s see how we can frame it without getting into trouble and stepping on toes of the people who are censoring us.

SPENCER: Right. Okay, so let’s start by saying that neither Martin nor I are physicians, and nothing we’re saying in this video should be construed as medical advice or for the treatment or diagnosis or therapy for any disease. Right. Now, having said that, we can also have intelligent conversations as free and sovereign human beings who are responsible for our own health.

MARTIN: Yeah, we can talk about physiology. We can educate about nutrition. We can educate about how things work in the body. We’re not promising that we are going to cure or remove or mitigate or whatever, any health condition. But we can talk to you about how health is built.

SPENCER: Having said that, as of pre-COVID days, one out of three people died of growths in their body.

MARTIN: Runaway cells.

SPENCER: Runaway cells. Tissue growing in ways we don’t want. Since vaccination, those numbers have increased. Some of them have. Sometimes people will go from diagnosis to death within one month. I suspect that in the decades to follow, it’ll be closer to 50, 60 or 70% of people will find that the end of their life is a result of growths in their body they don’t want. 

MARTIN: And post mRNA technology people now have this turbo version of this stuff going.

SPENCER: The term I’m going to use for this video is called Feral tissue. F-e-r -a-l. Now I’m using that word very specifically. And let me give you a way of, the reason is because the word that’s typically used has a lot of baggage associated with it. A lot of fear, a lot of scientific baggage. That word when someone hears it, they could feel that their body has betrayed them, is fighting them, is hurting them. It’s out to get them, that it’s an evil and malignant entity. And I think that’s the wrong way to think about our bodies. 

MARTIN: Yeah, let me just put it in my thoughts. The feral or feral or feral has to do with something that previously was civilized has gone wild. Think of it in that way.

SPENCER: So let’s say you have a family dog, a German shepherd, for instance. Right. And it’s a great dog and loves you and protects you and protects your family. But you weren’t properly trained on what it is to be a dog owner, and you don’t feed it for a month straight. Well, then at the end of the month, you hear a ruckus at three in the morning, you go outside and there’s your German Shepherd pinning down a chicken in your chicken coop that he broke into, and he’s about to eat it. Well, you go over there and you scold the dog and you try to take the chicken away, and he snarls at you, he may even bite you. Well, whose fault is that? It’s not the fault of the dog. It’s the fault of the owner who didn’t understand that you have to feed your dog. So, you could shoot the dog right then and there, but that would be violent and wasteful. You could poison the dog. Okay. Or you could go back inside and go get some raw hamburger meat and some bacon and a raw egg and put it in a bowl and bring it out and put it in front of your German shepherd. That went feral and went wild. And the dog might look at you and the chicken and the bowl of food and think for a second, and then sit down and gobble down all that food you made for it. Then lick your hand and go to sleep by your feet. You have now recovered your family dog. It is no longer feral. Right. That’s what we want to do with growths in the body. This was originally us and it went feral. It went a little wild and it doesn’t want to be wild. It’s just doing that, I believe, because we didn’t understand what it needed. So let’s back up. And what I want to show you Martin, if you’ll cue that logo now. Yeah.

MARTIN: I’m going to have on screen the logo. It has three circles and a bunch of words around it.

SPENCER: Right. So this logo is, let me pull it up here so I can look at it with you. Okay. So this logo is from the website feraltissue.com that will go into much more detail on the model that I’m presenting. And I want to express that this is a theoretical model at this point. We will have more information in a few months as we go through our first trials. Right now this is theoretical, but I’m going to present it to you because I think that there’s value to it, if only from a peer review perspective of opening up conversation. But also this may well be the model that is able to help a lot of people. So let’s talk about the model. 

On the logo, you see three circles and seven terms around. And the term at the bottom is good health. That’s where we want to be. That’s where we start. And at the end of the journey, that’s where we want to come back to. And then each of those three circles represents a trigger event, something that triggers the body into becoming feral or tissue thereof. The first one is SFCA, that’s a short chain fatty acids. And it’s brown because to remind us that that’s the color of stool, you need a healthy microbiome to make short chain fatty acids. And the other two we will get to that’s the AHR and the pH. We’ll get to those in a minute.

And then these six phrases around the, as you go around the circle, these are the various programs that the body, I believe, activates as a result of these insults, these mistakes that we’ve made. So the goal is to reverse these six programs, and we do that by addressing the programs directly, but by also dealing with the three causes in the middle that started them all. So let us start with the first one, and that would be short chain fatty acids. 

The microbiome, your gut, if it’s healthy, will make good short chain fatty acids including lactate, butyrate, propionate, acetate, valerate. These are all things that are very good for our health, and they control an enormous amount of our functioning. Now, you’ve heard of the term epigenetic. That’s the way of saying that, hey, genes are one thing, but the genes have to be turned on or off by things in our environment. And it is these epigenetic triggers that are as important as your genes, because a person can have terrible genes, but if they’re not activated epigenetically, that’s fine. And a person can have great genes, but if they’re not activated properly, then they don’t function properly. Even a good gene operated improperly is a bad outcome.

MARTIN: Yeah, you could start as a strong person and live a bad life and you’ll have bad outcomes. You can start as a weak person, but if you live well, you’re going to actually end up living a better quality of life than the person with great gifts who has abused them.

SPENCER: So let’s talk about what happens then. If the person doesn’t have enough short chain fatty acids because like most of us, they’ve taken antibiotics and glyphosate pesticides in their food and chlorinated water and so forth and so on. The body has less short chain fatty acids. Now, what the short chain fatty acids are supposed to do is they’re supposed to go on to these receptors that take them into the cell, and then they are there to interface with the nucleus and the DNA and give instructions to the DNA. Now, I want you to imagine that you’re a fisherman. Imagine a fisherman who needs to feed his family with five fish a day. He’s got a bunch of kids, and so he goes out to his favorite fish fishing spot every day, and he catches his five fish and feeds his family. Well, one day he goes out there, and then there’s only four fish and then three fish, then two fish. And now he’s down to one fish a day and his family’s getting hungry. So the next time he goes out, he dives off his boat and he looks around underwater and he goes, wow, there’s just not a lot of fish here. There are some fish, but not many.

SPENCER: So he goes back home and thinks about it. Makes four more fishing poles. Goes out the next day, puts all five fishing poles out in his boat, and now he’s getting some more fish. And it’s fun, right? So the fish in this analogy are the short chain fatty acids, and the fishing poles are the receptors that will pull in the short chain fatty acids into them, into the nucleus. Now, what the body will do when it doesn’t have enough of a particular ingredient that it needs, is to increase the number of fishing poles or increase the number of receptors. So that takes us to the first program, a receptor upregulation. It’s making more receptors to try to grab what it needs in an environment that is deficient. And so the indigo colored circle has the letters A, H, R. That stands for ARYL hydrocarbon receptor. That’s the receptors that we’ll be bringing in the short chain fatty acids. So let me tell you a story about a scientific mistake that happened 50 odd years ago that set us on the path of why I think feral tissue is still not resolved, medically speaking.

MARTIN: Well, it’s misunderstood because the genetic seduction happened, right? 

SPENCER: So two things happened. I mean, more than two of them.

MARTIN: Yeah. Right.

SPENCER: So back in the 70s, somebody found a new receptor in the body that would bind to dioxin, which is the chemical in Agent Orange. Incredibly toxic for the body. And they said, okay, this binds to dioxin. We’ll call this a hydrocarbon receptor, because dioxin is a hydrocarbon. Well, that’s stupid because God didn’t give us a hydrocarbon receptor, right? The first exposure of hydrocarbons for humans was maybe 800 AD in China, with the first oil well dug. Before that, we didn’t have hydrocarbons. So why would we evolve a receptor for hydrocarbons? Well, it was lazy. It was scientifically lazy. What they should have done is say, hey, this receptor is binding to hydrocarbons. Let’s find out what it’s really for. And they might have found out it’s for short chain fatty acids, but they called it a hydrocarbon receptor. And then they said, wow. This hydrocarbon, when it attaches to this hydrocarbon receptor, causes growths in the body that can kill people, causes feral tissue. And so everybody starts looking for toxins, like hydrocarbons that will cause that. Now, the other thing they found with feral tissue is there’s a lot of genetic damage. And everybody said, oh, look, there’s 50,000 genes that are screwy in people that have feral tissue. Certainly, somewhere something in there must be the key. There must be some gene that’s doing it. Okay, so these are the two errors. The idea that genetics are causing this, I think, is backwards. I don’t think that the genes are causing feral tissue. I think the feral tissue is causing the damage to the genes, and so they’re looking in the wrong place. Yeah.

MARTIN: Exactly right. This is the epigenetic explanation of what’s really going on.

SPENCER: Right. Now, that’s not to say that there aren’t some genetic mutations that make people more likely to have feral tissue, but more likely to if the short chain fatty acids aren’t there. More likely, if the toxins are there, it still requires some trigger, right? It’s a predisposition, but it still requires a trigger. So the first thing I would say is it’s not necessarily genetic. And the proof for this would be a landmark study that was done where they took the nucleus of a feral cell and put it in a healthy cell. And the healthy cell did not become feral.

MARTIN: Yeah,. You’re now discussing Thomas Seyfried and his research and he’s proven that this whole thing is a metabolic problem, not a genetic problem.

SPENCER: Right. And the other part of that study is they took a normal nucleus and put it in a cell that was feral, and it stayed feral because it wasn’t the DNA that was running the show for the feral issue, it was the environment the cell was in. 

MARTIN: And just to illustrate, I want to illustrate it so clearly, when you have an aquarium in which you are seeing sick fish, you don’t treat the fish, you change the water.

SPENCER: Yeah. Great point, great one. So because we’ve been looking at genetics as a prime mover rather than a secondary effect, And because we’ve been looking at hydrocarbons rather than because we’ve been calling these receptors hydrocarbon receptors rather than what they really are, which is short chain fatty acid receptors which can bind to hydrocarbons. The last 50 years, research went, in my opinion, down a very unfruitful path. 

MARTIN: Oh, totally. We’ve wasted billions of dollars. Sorry to keep interrupting. We’ve wasted billions of billions of dollars in the war on that. That is just so unfruitful.

SPENCER: Well, and hundreds and hundreds of millions of people have died.

MARTIN: Oh, yeah. We saved no one. 

SPENCER: So with all those extra hydrocarbon receptors, it’s just a vacuum sucking in these toxic metabolites of virus fungi, bacteria and parasites and these toxic chemicals that are causing feral tissue. So the first thing, one other thing to understand is this isn’t theory. 20% of all feral tissues are associated with viruses like Epstein-Barr, Cytomegalovirus, herpes virus that a lot of us are carrying. So, the spike protein Covid, actually hijacks the hydrocarbon receptor. It’s why it accelerates feral tissue so aggressively. So what do we want to do? Well, the first thing we want to do is at the very bottom, is we want to increase the short chain fatty acids. We can do that immediately by actually taking short chain fatty acids. We have a short chain fatty acid product called Phylamet. But also long term we want to recover the microbiome. And you know we’ve got protocols for that. But that’s not necessarily going to be enough once this process is started. So the next thing we need to do is deal with the hydrocarbon receptors. So the reason it is indigo-colored is because the ingredient that I think we should use for the hydrocarbon receptors is an extract of the indigo plant. Now, dioxin, for instance, has an incredibly strong bond to the hydrocarbon receptor.

SPENCER: It’s got like a, I don’t know, like a five-day half-life, something like that. You need to displace it. So what we want to do is we want to knock the toxins off the hydrocarbon receptors so they can’t cause problems. And then we want to pull them out and detoxify them. But to knock them off, you need something with a stronger bond than the toxin itself. And dioxin has an incredibly strong bond to this. So the one thing, the only thing I found that will actually make a stronger bond to the hydrocarbon receptor than some of these toxins is the indigo extract. And thankfully it’s nontoxic. So we have a product called Receptimet that has an indigo extract in it that I like. And what I want to do is I want to clean off the receptors, and I want to kind of seal them up temporarily so that while we’re pulling these toxins out of the body, while we’re working on supporting the immune system, to have the body deal with the viruses and fungi and bacteria and parasites, that those metabolites don’t hijack our hydrocarbon receptor systems. So I want to pull them. So, that’s the indigo color.

SPENCER: Well, I would actually say it’s more active than passive, right? I would say that these are actually little arms sticking out of the cells, grabbing and pulling things in. So all those receptors are actively looking for short chain fatty acids. They’re hungry for them. They want them and they grab on to what they think is short chain fatty acid. And it turns out it’s a toxic chemical. Some instructions by a bacteria or virus or a fungi to tell the DNA to stand, to tell the DNA, to tell the immune system to stand down. So it’s not so much it’s an opening in the cells. They’re actively being pulled in. So what we want to do is imagine you’ve got all these little hands pulling in all these toxins. What you want to do is you want to go and kind of knock the toxin off the hand and then put something else in that hand to keep it busy so it doesn’t do it again. And that’s what I like to use in indigo extracts for.

MARTIN: Awesome.

SPENCER: And when I looked up these indigo extracts in medicine, guess what I found. They’ve been used for thousands of years for guess what? Feral tissue. So I wasn’t the first person to figure this out. I was just the first person to figure out how it’s happening, right? We understood verbally for thousands of years that, hey, this works. They use it in Chinese medicine for feral tissue. They just didn’t have the biochemical sophistication at the cellular level a thousand years ago to understand why it was working, but they figured it out. God bless them. 

Okay, so this takes us now to the next of the programs, which is fermentation. So let’s say that we didn’t fix the microbiome. The short chain fatty acids are deficient. And now the hydrocarbon receptors are increasing. And that’s still not enough. The body says I still even though I’ve made all these extra receptors, I’m still not getting enough short chain fatty acids, but I really need them. So here’s my theory. My theory is that the body will then get or shift some cells from their normal function to actually making short chain fatty acids, that it’ll make some cells in the body ferment like the microbiome. Now, if you look at the work of Otto Warburg and you ask, what Otto Warburg said about feral tissue is he said, these are fermenting cells. Well, what’s the end product of fermentation? Well, in the gut, the end product of fermentation is short chain fatty acids. Now, what are the,

MARTIN: Lactic acid more often than anything else. Right?

SPENCER: Well, lactic acid is the one we’ve looked at. So let’s talk about that. So feral tissue has a lot of strange behavior that doesn’t make sense unless you are outside of this model. One thing is, it makes a lot of lactic acid or lactate. But that’s a very inefficient way to make energy. It’s like 16 times less efficient in making energy. And so people say, well, feral tissue is stupid. It’s bad. It’s dysfunctional because it’s not making energy properly. And if you look at it from the model of that tissue is designed to make energy. You’re right. It’s not very efficient, which is why it has to run 10 to 100 times faster. However, if you look at it from the model of it’s trying to make short chain fatty acids for you, it’s wonderfully efficient. So it’s very good from that perspective that it’s doing right. So the question is, do you think the body is stupid? If you think it’s stupid, then it’s making a mistake and very inefficient. But if you think it’s smart and it’s doing it on purpose for what it thinks is a temporary scenario, it thinks, hey, maybe the person got injured and you know, the caveman got bitten by a saber tooth tiger and isn’t eating food for 2 or 3 weeks by the fireside while he recovers.

SPENCER: That person’s not going to get food to make short chain fatty acids.  The body does short term things that have long term consequences because it, I’m not going to say the body is omnipotent. It’s very intelligent. But it does have this flaw that sometimes it will do things in the short term that will have a long term consequence. Now from the intelligence of the body, if you’re 20 and it gets you to live to 40 before you die, or 30 before you die, and you get to have a bunch of kids, that’s success. Your genes have passed on, right? Your DNA is fine with you having a lot of kids that you have at 20 raise till they’re old enough to be on their own and they’re 30, and then you pass away. So if we say, well, the body is stupid, it’s making these short term benefits for long term problems. Yeah. But from its perspective it only wants it only cares if you make it to 40. Right. Anything after that? Well, maybe you’re out of the gene pool at that point. You’ve already passed it on. So even to say,

SPENCER: It’s still not stupid. It’s doing what it wants from its perspective of passing on its genetics. It’s just not doing what we want for, say, living to 80 and good health. So okay, so now we’ve got these cells that are making lots of lactate. Well, what about the other short chain fatty acids? Acetate, propionate, butyrate. That’s an hour long conversation that if you go to feral tissue com there’s a video that shows the biochemistry about how all the mechanics and enzymes of making all those other short chain fatty acids are increased in feral tissue. Let’s just say that you know that science is there, whether it’ll be proven out at peer review levels, we don’t know. But there is definitely some good scientific justification for suggesting that feral tissue is there primarily to make short chain fatty acids.

MARTIN: So I guess the bottom line for me is let’s not be so simplistic as to think that it’s some kind of a mistake that makes lactic acid by accident.

SPENCER: Yeah, yeah. Or any of the other short chain fatty acids. Right. All right. And some people will say, oh, well, lactic acid increases feral tissue. Well, there is a study that said, okay, is it the lactic acid or is it the acid. And what they did is they added lactic acid and some buffer agents. So it wasn’t acidic anymore. It was neutral to some feral tissue. And the feral tissue started behaving normally. So actually short chain fatty acids seem to have an anti feral tissue effect, but it’s the acids that caused the problem. So why does that happen? So think about where acids are meant to be formed. Short chain fatty acids are meant to be formed in the large intestine. And the large intestine has several defenses against this. One is every four days the cells of the large intestine will slough off and go out into the feces that’s there so that they won’t become feral, because if you leave a cell around acid long enough, it can become feral, it can become damaged. So it’ll slough off. But one of the first, the other thing is it creates a lot of mucus as a protection. And we’re going to get to both of those. But one of the first things that happens, first things that happens, the green pH circle, it starts dropping the pH in the area. Now feral tissue is interesting. It’s actually alkaline on the inside and acid on the outside.

SPENCER: And again that’s not something we can necessarily get into at this point in this video. I would watch the other videos for that. But the green circle with the pH is there to tell us that it’s pH. That’s the final driver of the worst of the problems. And the green color is there because if you check your urine, you want the pH paper to turn green, which suggests that you’re alkaline enough. So that’s what that color stands for. So the first thing that happens when tissue gets damaged is stem cells are recruited to repair tissue. Now, if you look at feral tissue, it de-differentiates. It doesn’t look like the tissue it started. If it starts as breast cells, it doesn’t look like feral tissue in the breast. Doesn’t look like a breast anymore. Feral tissues in the lung doesn’t look like lungs anymore, and the prostate doesn’t look like a prostate. It differentiates. It’s becoming more and more like a stem cell. Well, okay. That’s how injuries are repaired. The body takes normal cells and pushes them back towards stem cells. That differentiates them. So the beginning process of feral tissue is a healing process. It’s the creation of stem cells. And the problem is that those stem cells can’t heal the injury caused by the acids that are building up in the tissue. So the body is trying, but it doesn’t really have the capacity to do that. 

MARTIN: This is an interesting point here because there are some other scientists that talk about electricity. Doctor Tennant came up with that, and he’s showing that cells to repair themselves correctly need to be in an alkaline environment. Right. So we’ll talk about 50 millivolts and all that.

SPENCER: Yeah. We’ll talk about the voltage in about another ten minutes.

SPENCER: So the stem cell part of this program is actually a healing response of the body. So the fermentation, and all of these things, the upregulation is the body trying to deal with short chain fatty acids, the fermentation, the lack of short chain fatty acids, the stem cell aspect and dealing with the injury. So the next thing that happens is mucus is produced or mucus. Feral cells have a lot of mucus in them. And well, where in the body do you find mucus? A lot of it you find in the large intestine as a protection against acids, or in the stomach as a protection against stomach acids. So the mucus is explained by this. So then the next thing that happens is sloughing. Now in the large intestine, when the cells are about four days old, they slough off so they don’t get damaged so that the damage they’ve incurred from the short chain fatty acids doesn’t cause them to become problematic. Where does a cell sluff off if it’s in the breast or the prostate? It can’t go anywhere. Well, actually it can, and it can leave and go into the blood vessels of the lymph and set up shop somewhere else. So sloughing is a process of the escape of those cells to other parts of the body.

MARTIN: I guess proliferation would be the word for that?

SPENCER: So the last program we’re going to get is called the trophoblast program. And that becomes a part of the placenta. Now, in animals other than humans that have an egg yolk sac. Reptiles and birds, part of the placenta actually leaves and goes to become the egg yolk. And it does the same for humans. But since we don’t make egg yolks, it then decides to come back and on its way back, it passes through all the tissue and tries to make its way back to the genital crest and into the ovaries and the testes. Because those are germ cells, but not all of them make the return journey. And it’s these kinds of lost trophoblastic cells that we believe are the seeds for future feral tissue. So we all have these seeds in us, places where a couple of hundred of these cells throughout the body that didn’t make it back. And these are the ones that I believe are recruited by the body to make short chain fatty acids for us because they’re stem cells. They’re they’re pluripotent. They can become anything.

SPENCER: So they said, okay, well become this. All right. So let’s take a look at some very interesting equivalence between the trophoblast or the placenta and feral tissue. Both grow very quickly. They both can exist in an environment of low oxygen and high carbon dioxide. They both have high blood, accelerated blood vessel growth. They both lack contact inhibition. That is, they don’t stop growing when they touch each other. So let’s say I get a paper cut. Well, the cells on either side of the paper cut are going to grow until they meet in the middle. And then when they touch each other, that’s contact inhibition. They say, okay, I should stop growing now. Placenta and feral cells don’t do that. They keep growing. Both have limited Krebs cell activity and make lactic acid. They both create the pregnancy hormone hCG, which triggers progesterone, which then stimulates migration and evasive behavior. You can actually test for feral tissue with a pregnancy hormone. So that’s telling you that there’s something going on between pregnancy placentas an